mist |
metabolite in safety testing |
baloff |
mit insaf |
mitral insufficiency |
|
mitochondria |
A structural unit within cells that produces energy through a process called cellular respiration. |
|
mitochondria |
A structural unit within cells that produces energy through a process called cellular respiration. |
|
mitochondrial toxicity |
Damage to mitochondria. Mitochondrial toxicity may affect different parts of the body, including the heart, nerves, muscles, pancreas, kidneys, and liver. Conditions resulting from mitochondrial toxicity can include muscle weakness, inflammation of the pancreas (pancreatitis) high levels of lactic acid in the blood (lactic acidosis) changes in distribution and amount of body fat (lipodystrophy) and fatty liver (hepatic steatosis) Use of certain antiretroviral (ARV) drugs may cause mitochondrial toxicity. |
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mitochondrial toxicity |
Damage to mitochondria. Mitochondrial toxicity may affect different parts of the body, including the heart, nerves, muscles, pancreas, kidneys, and liver. Conditions resulting from mitochondrial toxicity can include muscle weakness, inflammation of the pancreas (pancreatitis) high levels of lactic acid in the blood (lactic acidosis) changes in distribution and amount of body fat (lipodystrophy) and fatty liver (hepatic steatosis) Use of certain antiretroviral (ARV) drugs may cause mitochondrial toxicity. |
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mit insaf |
mitral insufficiency |
|
ms |
mitral stenosis |
|
mixed tropic virus |
A strain of HIV that can enter and infect a host cell by binding to either the CCR5 or CXCR4 receptor on the host cell. To enter a host cell, HIV must first attach to a CD4 receptor, then attach to either the CCR5 or CXCR4 receptor, and finally fuse its membrane with the host cell membrane. HIV is usually R5-tropic (uses CCR5) during the early stages of infection, but the virus may later switch to using either only CXCR4 or both CCR5 and CXCR4. |
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mixed tropic virus |
A strain of HIV that can enter and infect a host cell by binding to either the CCR5 or CXCR4 receptor on the host cell. To enter a host cell, HIV must first attach to a CD4 receptor, then attach to either the CCR5 or CXCR4 receptor, and finally fuse its membrane with the host cell membrane. HIV is usually R5-tropic (uses CCR5) during the early stages of infection, but the virus may later switch to using either only CXCR4 or both CCR5 and CXCR4. |
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mld |
minimum lethal dose |
|
mlg |
medical labeling group |
oxana135 |
mMIDI |
modified Minnesota Impulsive Disorders Interview |
ННатальЯ |
mn. |
mandible |
Ying |
Mo |
C17H19O3N |
MichaelBurov |
MoA |
Mechanism of Action |
Dimpassy |
mobile health |
m-health |
MichaelBurov |
mobile health |
mHealth |
MichaelBurov |
micu |
mobile intensive care units |
alumna1979 |
MoCA (test) |
Montreal Cognitive Assessment |
Val_Ships |